Jiazhen M Dai

Surveillance de l'innocuité des médicaments sur ordonnance après commercialisation.

Santé Canada

Principales publications

  1. Dai JM, et al. A comparative study of α-adrenergic receptor mediated Ca2+ signals and contraction in intact human and mouse vascular smooth muscle. Eur J Pharmacol. 2010 Mar 10;629(1-3):82-8.

  2. Dai J, et al. Endothelin-1 mediated waves-like [Ca2+]i oscillations in intact rabbit inferior vena cava. J Vasc Res. 2007; 44(6): 495-503.

  3. Dai JM, et al.  Acetylcholine-induced asynchronous calcium waves in intact human bronchial muscle bundle. Am J Respir Cell Mol Biol. 2007; 36(5): 600-608.

  4. Dai JM, et al.  The mechanism of ACh-induced asynchronous calcum waves and tonic contraction in the porcine tracheal muscle bundle. Am J Physiol Lung Cell Mol Physiol. 2006; 290(3): L459-69.

  5. Dai J, et al.  Rearrangement of the close contact between the mitochondria and the sarcoplasmic reticulum in airway smooth muscle. Cell Calcium. 2005; 37(4): 333-40.

  6. Chen JH, Riazy M, Wang SW, Dai JM, et al. Sphingosine kinase regulates oxidized low density lipoprotein mediated calcium oscillations and macrophage survival. J Lipid Res. 2010 May;51(5):991-8.

  7. Kuo KH*, Dai J*, et al. The relationship between asynchronous Ca2+ waves and force development in intact smooth muscle bundles of the porcine trachea. Am J Physiol Lung Cell Mol Physiol. 2003; 285(6): L1345-53. *contributed equally.

  8. Lee CH, Kuo KH, Dai J, van Breemen C.Asynchronous calcium waves in smooth muscle cells. Can. J. Physiol. Pharmacol. 2005; 83: 1-9.

  9. Lee CH, Kuo KH, Dai J, et al.Calyculin-A disrupts subplasmalemmal junction and recurring Ca2+ waves in vascular smooth muscle. Cell Calcium. 2005; 37(1): 9-16.

  10. Poburko D, Kuo KH, Dai J, Lee CH, van Breemen C. Organellar junctions promote targeted Ca2+ signaling in smooth muscle: Why two membranes are better than one. Trends Pharmacol Sci. 2004; 25(1): 8-15.

  11. McGuire JJ, Dai J, et al. Proteinase-activated receptor-2 (PAR2): vascular effects of a PAR2-derived activating peptide via a receptor different than PAR2. J Pharmacol Exp Ther. 2002 Dec;303(3):985-92.